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In patients requiring insulin therapy, the ideal frequency of glucose monitoring has not been established. A common practice is to check the glucose level four times daily. A first morning glucose level can rule out fasting hyperglycemia, and additional one- or two-hour postprandial values can ensure adequate control. Postprandial testing is preferable to preprandial testing. In one randomized study comparing postprandial and preprandial blood glucose monitoring in patients with gestational diabetes who required insulin therapy, those who measured their glucose levels after meals had larger drops in A1c -3.0 versus -0.6 percent, P .001 ; , gave birth to infants with lower birth weights 3, 469 g [7 lb, 10 oz] versus 3, 848 g [8 lb, 7 oz], P .01 ; , and had fewer cesarean deliveries 12 versus 42 percent, P .04 ; .19 [Evidence level B, lower quality RCT] There is neither objective evidence nor a clinical guideline to support a frequency for glucose monitoring in patients with diet-controlled gestational diabetes. In these patients, an acceptable practice is to use the four-times-aday schedule on two days per week and begin more intensive treatment if two values per week exceed the limits.
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Ized on cultured retinal pigmented epithelial and ciliary muscle cells from cynomolgus monkey eyes, thereby suggesting that PGF2 analogues decrease IOP by increasing uveoscleral outflow in both monkeys [25, 41-43] and humans [14, 44]. Additionally, recent studies demonstrate the presence of all prostanoid receptors in the human trabecular meshwork, suggesting a potential role for prostanoid receptor agonists and antagonist on the outflow of aqueous humor through the trabecular meshwork for the management of glaucoma [45]. There is increasing evidence in the literature to suggest that both PGE2 and PGI2 exhibit neuroprotective roles in vitro [4649]. However, extensive research is still required both in vitro and in vivo to confirm this function. Further biochemical, molecular and pharmacological studies are required in order to elucidate both mechanistic and functional details of ocular prostanoid receptors. Such knowledge may then provide significant insight to aid the design of new therapeutic agents for use in IOP reduction and or novel treatment strategies for glaucoma such as neuroprotection and enhanced blood flow. Additionally, based on the widespread distribution of ocular prostanoid receptor mRNA and limited functional evidence, one might speculate a potential role for prostanoid-based medications in the treatment of ocular allergies and inflammation. ACKNOWLEDGEMENTS This work was supported by the J. P. Bickell Foundation and the E. A. Baker Foundation for the Prevention of Blindness. We thank Alcon Research Ltd for previous financial support and intellectual collaboration. We thank Dr. Melanie Campbell for the generous donation of human ocular globes and Inka Tertinegg for assisting in the collection of human eyes. Lastly, we thank Dr. D. J. Crankshaw for his assistance in the collection of human myometrial tissue. REFERENCES.
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1. Young NS. Acquired aplastic anemia. Ann.Intern.Med. 2002; 136: 534-546. Young NS. Drugs and chemicals. In: Young NS, Alter BP, eds. Aplastic Anemia, Acquired and Inherited. Philadelphia: W.B. Saunders; 1994: 100-132. 3. Young NS. Viruses as agents of marrow failure. In: Young NS, Alter BP, eds. Aplastic Anemia, Acquired and Inherited. Philadelphia: W.B. Saunders; 1994: 133-158. 4. Choudhry VP, Gupta S, Gupta M, Kashyap R, Saxena R. Pregnancy associated aplastic anemia - a series of 10 cases with review of literature. Hematolgy 2002; 7: 233-238. Cartwright RA, McKinney PA, Williams L et al. Aplastic anaemic incidence in parts of the United Kingdom in 1985. Leuk.Res. 1988; 12: 459-463. Linet MS, Tielsch JM, Markowitz JA et al. An apparent cluster of aplastic anemia in a small population of teenagers. Arch.Intern.Med. 1985; 145: 635-640. Morgan GJ, Palmer SR, Onions D et al. A cluster of three cases of aplastic anaemia in children. Clin.Lab.Haematol. 1988; 10: 29-32. Whang KS. Aplastic anemia in Korea: a clinical study of 309 cases. In: Hibino S, Takaku F, Shahidi NT, eds. Aplastic Anemia. Baltimore: University Park Press; 1978: 225-242. 9. Bernard J. Esquisse d'une hmatologie gographique. Nouv.Rev .Hematol. 1963; 3: 51-58.
Studies have also reported many days of antioxidant decline [376] or a complete lack of recovery at 8 days after trauma [18]. The pattern of change in antioxidant levels was similar in all patients irrespective of trauma severity or premorbid medical condition, a feature observed by other studies [18, 376].
37. Hesbacher P, Rickels K, Downing RW, Stepansky P. Assessment of psychiatric illness severity by family physicians. Social Science & Medicine 1978; 12 1-A ; : 45-47. 38. Hesbacher PT, Rickels K, Morris RJ, Newman H, Rosenfeld H. Psychiatric illness in family practice. J Clin Psychiat 1980; 41: 6-10. Derogatis LR, Melisaratos N. The brief symptom inventory: an introductory report. Psychol Med 1983; 13: 595-605. Bolton P. Cross-cultural validity and reliability testing of a standard psychiatric assessment instrument without a gold standard. J Nerv Ment Dis 2001; 189: 238-242. Rabin R, de Charro F. EQ-5D: a measure of health status from the EuroQol Group. Ann Med 2001; 33: 337-343. Hillner BE, Smith TJ. Cost-effectiveness analysis of three regimens using vinorelbine Navelbine ; for nonsmall cell lung cancer. Semin Oncol 1996; 23 2 Suppl 5 ; : 25-30. 43. Konski A, Scott C, Movsas B, et al. Cost-utility analysis of various treatments for non-small cell carcinoma of the lung. 2000; The Second European Conference on the Economics of Cancer, Brussels Abstract ; . 44. Schulz MW, Chen J, Woo HH, et al. A comparison of techniques for eliciting patient preferences in patients with benign prostatic hyperplasia. J Urol 2002; 168: 155-159. Badia X, Herdman M, Kind P.The influence of ill-health experience on the valuation of health. Pharmacoeconomics 1998; 13: 687-696. Wu AW, Jacobson KD, Frick DL, et al. Validity and responsiveness of the EQ5D as a measure of healthrelated quality of life in people enrolled in an AIDS clinical trial. Qual Life Res 2002; 11: 273-282. Glasziou PP, Simes RJ, Gelber RD. Quality adjusted survival analysis. Stat Med 1990; 9: 1256-1276. Zelen M. The randomization and stratification of patients to clinical trials. J Chron Dis 1974; 27: 365-375. Kupelian PA, Elshaikh M, Reddy CA, et al. Comparison of the efficacy of local therapies for localized prostate cancer in the prostate-specific antigen era: a large single-institution experience with radical prostatectomy and external-beam radiotherapy. J Clin Oncol 2002; 20: 3376-3385. Schoenfeld D. Sample-size formula for the proportional-hazards regression model. Biometrics 1983; 39: 499-503. Lan KKG, DeMets ER. Discrete sequential boundaries for clinical trials. Biometrika 1983; 70: 659-663. O'Brien PC, Fleming TR. A multiple testing procedure for clinical trials. Biometrics 1979; 35: 549-556. Freidlin B, Korn EL. A comment on futility monitoring. Controlled Clinical Trials 2002; 23: 355-366. Kaplan EL, Meier P. Nonparameteric estimation from incomplete observations. J Amer Statist Assoc 1958; 53: 457-481. Cox DR. Regression models and life tables. J Royal Stat Soc 1972; Series B ; 34: 187-229. 56. Freidlin B, Korn EL. Testing treatments effects in the presence of competing risks. Stat Med 2004; 24: 1703-1712. Mantel N. Evaluation of survival data and two new rank order statistics arising in its consideration. Cancer Chemother Rep 1966; 50: 163-170.
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