The UK Patents and Designs Journal No. 6100 ; reports the filing of two SPC applications from Novo Nordisk that were reported 3 weeks ago in Current Patents Gazette 0612. These filings both relate to EP705275, which claims ASP-B28 insulin crystals comprising insulin aspart and protamine. Novo Nordisk already has a granted SPC for EP705275, relating to a ratio of 30% free, rapid-acting and 70% protamine-cystallized insulin aspart NovoMix 30 ; that can be administered using Novo Nordisk's FlexPen insulin injection device. This granted SPC is due to expire on 22nd June 2015. The new filings relate to suspensions comprising insulin aspart and protamine in 70 30 NovoMix 70 ; and 50 NovoMix 50 ; ratios having an increased percentage of rapid-acting insulin, raised from 30% to 70% and 50% respectively. These new suspensions make it possible to intensify insulin treatment for diabetes over time. If granted, these SPCs are due to expire on 19th June 2019. The PDJ also reports the entering into force of SPC GB01 045 on 26th March 2006, covering nateglinide. This SPC is based on EP0196222 assigned to Ajinomoto and is due to expire on 25th March 2011. Nategliide Starlix ; , an oral hypoglycemic agent for the treatment of type II diabetes that acts through rapid, shortterm stimulation of insulin production, was codeveloped with Yamanouchi and Morishita in Japan. In 2004, Japanese sales of nateglinide by Yamanouchi totaled $43.1 million, representing a year-on-year sales growth of 9.9%. Ajinomoto granted Novartis exclusive development and marketing rights outside South East Asia, except for the UK and the Republic of Ireland, and by December 2000, Novartis had launched the product in Switzerland and Brazil. By 2001, nateglinide had also been launched in the US, Germany and the UK. In October 2000, Novartis and Merck KGaA announced that nateglinide had been approved by Swiss regulatory authorities for the treatment of type II diabetes, alone or in combination with Merck's metformin. SPC GB93 110 granted to Astra Draco for its long-acting beta-2 adrenoceptor agonist, bambuterol, is reported to have expired. The drug has been launched in a once-daily formulation Bambec ; for the relief of nighttime discomfort caused by asthma. The SPC came into force on 30th June 2001, giving bambuterol protection until 26th March 2006 based on its Danish marketing authorization. SPCs granted in other European countries have also expired, except the Swiss SPC, which is due to expire in June 2006. A year ago April 21, 2005 ; , the European Court of Justice gave their decision that Swiss Marketing Authorisations should be used as the first Community Approval date, if earlier than the EU Authorisation, because of their validity in Liechtenstein. However on June 1, 2005 Liechtenstein and Switzerland modified their bilateral agreement so that approvals in Switzerland would not immediately be valid in Liechtenstein but would be placed on a "negative list" until they were approved elsewhere in Europe or until one year had passed. Consequently, a first Swiss approval would not be relevant for calculation of any SPC expiry date for such products. To date, only 7 drugs have been added to the list, three of which were quickly removed due to an earlier European approval. A further one, covering Aptivus tipranavir ; marketed by Boehringer Ingelheim, was approved August 2005 in Switzerland, but was removed in November following EU approval in October 2005. As SPC applications filed in the UK and other countries cited both the EU and the earlier Swiss approval dates, it would have been interesting to see which date was used by the Patent Offices. However this becomes irrelevant as the SPC is limited to 5 years from patent expiry in this case May 2020 ; which is earlier than the date reached using either Swiss or EU approvals. Exjade deferasirox ; , Vidaza 5-azacitidine ; and Nexavar sorafenib ; remain on the list having been approved in Switzerland but not elsewhere in Europe. York Pharma plc has filed a UK initial application claiming derivatives of 18glycyrrhetinic acid, having one earlier international application published on the subject of antimycotic nail polish based on Bayer's abafungin. However, the company's website indicates a more extensive technology base in dermatologicals, referring to ownership of nine international patent families. Certainly the US equivalents of EP365915 have now been assigned by Bayer to York Pharma. Some medications may require prior authorization in order to be covered under your patient's pharmacy benefit plan. Some medications may also have limits on the quantities that can be covered. RegenceRx has implemented prior authorization and quantity level limits for select medications in order to promote their appropriate use, while deterring unproven or experimental usages. Medications Requiring Prior Authorization Non-formulary medications must be approved for members on closed formulary plans. We require that the member have tried and failed formulary alternatives first. Other medications requiring prior authorization include those that may be prone to overuse or present potential safety issues, medications that have limited uses based on scientific studies or FDA approval and medications that may be prescribed for conditions which are not covered or that require diagnostic tests to ensure medical benefit. Finally, prior authorization may also be required for some medications with less expensive options. A list of medications requiring prior authorization can be found at regencerx . This list also includes medications with set quantity limits, because glimepiride.

5. Khan SS, Xue JL, Kazmi WH, et al. Does predialysis nephrology care influence patient survival after initiation of dialysis? Kidney Int 2005; 67 3 ; : 1038-46. 6. Caskey FJ, Wordsworth S, Ben T, et al. Early referral and planned initiation of dialysis: what impact on quality of life? Nephrol Dial Transplantation 2003; 18 7 ; : 1330-8. Guideline CKD 3.2 Patients should have a functioning native arteriovenous fistula or a Tenckhoff catheter in place by the time that initiation of dialysis is required. Audit Measures: 1. Proportion of patients in whom a native arteriovenous fistula is used for the first chronic haemodialysis treatment. 2. Proportion of patients electing to have peritoneal dialysis, who start peritoneal dialysis without the need for temporary haemodialysis. Rationale: A native arteriovenous fistula AVF ; is widely regarded as the optimal form of vascular access for patients undergoing haemodialysis. The presence of a mature AVF at the time of first haemodialysis reduces patient stress and minimises the risk of morbidity associated with temporary vascular access placement as well as the risk of infection. Similarly, timely placement of a Tenckhoff catheter allows adequate training prior to the need for dialysis and avoids the need for temporary haemodialysis. Part 1 of the NSF for Renal Services recommends that patients should be referred for AVF formation at least 6 months prior to the anticipated date of initiation of haemodialysis and those opting for peritoneal dialysis should be referred for insertion of a Tenckhoff catheter at least 4 weeks prior to initiation of dialysis 1. Renal Units should collaborate with Surgical Services to set up a robust system that facilitates timely referral for and formation of vascular access or Tenckhoff catheter insertion. This should include a system for prioritising cases according to expected date of dialysis initiation. In addition, provision should be made for adequate bed and theatre-time availability to meet anticipated need. References 1. Department of Health. National Service Framework for Renal Services, Part 1: Dialysis and Transplantation. 2004.

Clamp the membrane potential and the intracellular concentrations of Na and DA, in contrast to studies with cell populations. AMPH is also able to depolarize hDAT cells Kahlig et al., 2004 ; , which could lead to an increase in intracellular Ca2 . Our results demonstrate that the increase in intracellular Ca2 promoted by AMPH is mediated by DAT and dependent upon thapsigargin-sensitive stores in suspended cells. Similar results were obtained using confocal imaging of single cells expressing the human norepinephrine transporter DiPace et al., 2003 ; . At low concentrations of AMPH, extracellular Ca2 might enter the HEK cells through voltage-dependent Ca2 channels Berjukow et al., 1996 ; or by Na Ca2 exchange Giambalvo, 2004 ; . The blockade by cocaine of this effect of AMPH delineates this response as DAT-mediated. Buffering intracellular Ca2 with BAPTA inhibited AMPH-induced DAT-mediated outward currents and DA efflux. Because intracellular BAPTA did not affect AMPHstimulated inward currents, it is unlikely that the BAPTAinduced reduction in DA efflux is a result of DAT trafficking. Buffering intracellular Ca2 also decreased the steep voltage dependence of DA efflux Fig. 2A ; , suggesting that the voltage sensitivity of reverse transport might rely on the presence of Ca2 . Increases in intracellular Ca2 in response to AMPH have also been reported by Mundorf et al. 1999 ; in bovine chromaffin cells and Chen et al. 1998 ; in the African snail Achatina fulica Ferussac. In the latter study, however, the concentration of AMPH used was very high 270 M ; , and no relationship to transport was demonstrated. In the chromaffin cell study Mundorf et al., 1999 ; , the source of cytoplasmic Ca2 in the AMPH response was demonstrated to be catecholaminergic vesicles. The mechanism was most probably attributable to a weak base action of AMPH because methylamine showed the same activity. In contrast, the hDAT cells used in the present study do not contain synaptic vesicles. That thapsigargin reduced both the ability of AMPH to elicit increases in intracellular Ca2 and AMPH-induced DA efflux suggests that the release of Ca2 from the endoplasmic reticulum is essential for DAT-mediated outward transport. Because thapsigargin alone raised intracellular Ca2 Fig. 5B ; but did not cause detectable DA efflux Fig. 6 ; , intracellular Ca2 by itself is not sufficient to cause DA efflux. The mechanism of internal Ca2 is currently unknown. Increases in internal Ca2 can lead to activation of protein kinase C independent of diacylglycerol production Trilivas and Brown, 1989 ; , so it is possible that the AMPH-induced increase in internal Ca2 could activate PKC. An increase in PKC activity because of AMPH-induced transporter activation has been demonstrated previously Iwata et al., 1997; Giambalvo, 1992, 2003 ; . Direct PKC activation by phorbol esters enhances DA efflux, whereas PKC inhibitors block AMPH-induced DA efflux from both rat striatum and PC-12 cells Giambalvo, 1992; Kantor and Gnegy, 1998; Cowell et al., 2000; Kantor et al., 2001 ; . Moreover, PKC activation leads to phosphorylation of N-terminal serines in DAT Foster et al., 2002 ; . We have demonstrated recently that Nterminal phosphorylation of DAT is essential for AMPHinduced DA efflux and currents Khoshbouei et al., 2004 ; , thereby providing a potential mechanistic link between internal Ca2 and efflux. In summary, our results allow us to propose the following, for instance, lincoln navigator.

When directed by the commanding general of I MEF, MWSS 372 and Blue Force METOC personnel will combine to form the JMFU. a ; U ; The JMFU will be established at Yuma, BLUELAND. b ; U ; MWSS 372's METMF, forward deployed to Auxiliary II, BLUELAND, will be the alternate JMFU. The alternate JMFU is responsible for producing and disseminating JMFU products: 1 ; When directed by the JMO. 2 ; If connectivity with the primary JMFU is lost for a period of six hours. 4 ; U ; MWSS 372 will provide METOC support to all forward-deployed units located in the vicinity of Auxiliary II, BLUELAND. 5 ; U ; When directed by the JMO, MWSS 372 will provide a fully equipped MEF weather support team, capable of rapid redeployment, to accompany operational commanders and to provide on-scene METOC services. 6 ; U ; The HQ AFWA and the NAVMETOCCOM will, through their centralized facilities, provide centralized products. 7 ; U ; The 55th Space Weather Squadron 55SXS ; will provide specialized space METOC products. 8 ; U ; Units at all echelons will follow the steps below to determine and fill METOC support requirements. Higher echelon units receiving shortfalls will look within their resources for the required capability. a ; U ; Determine METOC service support requirements. b ; U ; To the extent possible, provide resources from organic organizations to satisfy their requirements. c ; U ; When unable to satisfy their requirements, notify the next highest echelon of the shortfall. 9 ; U ; Intelligence units at all echelons of command will ensure that all TARWI and Forward Area Limited Observing Program FALOP ; inputs are passed to the JMFU in a timely manner. 10 ; U ; Pilot reports received by METOC ATC personnel will be forwarded to the JMFU in a timely manner. 11 ; U ; METOC reports include the following. Starlix was granted marketing approval by the european commission in april 200 this follows launches in brazil sept 00 ; , usa feb 01 ; and switzerland mar 01 ; , and starlix will be launched in many other countries around the world during 2001-200 starlix active ingredient: nateglinide starlix is available in film-coated tablets at three doses: 60 mg pink, round, bevelled edge tablets with starlix marked on one side and 60 on the other and viramune. Nateglinide [N- ; -D-phenylalanine] is a novel, highly physiologic, mealtime glucose regulator recently approved for the treatment of type II diabetes mellitus. Nateglinie has a rapid onset and short duration of insulinotropic action that results in reduction of mealtime glucose rise and lowers the postabsorptive potential for hypoglycemia in humans and experimental animals Fujitani et al., 1996; Hu et al., 1998; Ikenoue et al., 1997a, b; Karara et al., 1999; Hanefeld et al., 2000; Keilson et al., 2000 ; . In contrast to sulfonylureas, nateglinide increases pancreatic -cell sensitivity to ambient glucose without increasing basal insulin secretion Morimoto et al., 1998; Karara et al., 1999; Keilson et al., 2000 ; . Recently, the identification, synthesis, and pharmacologic testing of several nateglinide metabolites isolated from urine or bile of rat, dog, and human origin were reported Takesada et al., 1996; Mihara et al., 1997; Ono et al., 1997 ; . Only one of the identified metabolites designated M7 ; that possessed an unsaturation in the isopropyl side chain was found to have activity comparable with nateglinide. Do not take any other medicines without asking your doctor first and nicotine, for instance, novartis.
Clinical efficacy Two randomized, double-blind, placebo-controlled clinical trials formed the basis for the approval of P-O3FA monotherapy in conjunction with diet for the treatment of patients with severe hypertriglyceridemia i.e., 500 mg dL [5.65 mmol L] ; Table 1 ; .25, 26 In one trial, P-O3FA 4 g day was compared with placebo corn oil ; in 42 patients who had triglycerides of 500 mg dL 5.65 mmol L ; and 2000 mg dL 22.60 mmol L ; for 16 weeks.25 In 16 weeks, P-O3FA increased the serum concentration of phospholipid-bound EPA and DHA by twofold to threefold, from 1.4. The new insulin secretagogue nateglinide is an oral medication that effectively normalizes first-phase insulin response, thus controlling postprandial hyperglycemic excursions without causing a high rate of hypoglycemia and nortriptyline!

O If the beneficiary had no regular benefit days remaining when admitted, available lifetime reserve days are used automatically for each day of the stay. Exceptions exist if the beneficiary elects not to use them, or the charges for which the beneficiary is liable, if he does not use lifetime reserve days, do not exceed the charges for which he would be liable if he used them. Using lifetime reserve days he would be responsible for the sum of the coinsurance amounts for the lifetime reserve days that would be used plus the total charges for outlier days, if any, for which no lifetime reserve days are available. In the latter case the beneficiary will be deemed to have elected not to use any lifetime reserve days. ; An election by the beneficiary not to use lifetime reserve days applies to the entire stay and precludes any payment for the stay. A deemed election not to use lifetime reserve days applies to the entire stay and precludes any payment for the stay unless payment may be made under the guarantee of payment. The number of days for which utilization is charged may be different from the number used in PRICER to compute outlier status or the number of Medicare patient days shown on the cost report. 415.2 Determining Charges and Days to be Treated as Noncovered on the Bill.--The rules in subsections A, B, and C apply in determining the charges and days to be treated as noncovered for utilization. See 4l5.l for counting utilization days. ; Any days on which you did not furnish any services covered under Part A are not charged to utilization and are not counted as Medicare patient days. See 4l5.l4 for the effect of payment for noncovered services under waiver of liability or the grace day provision. See 4l5.l5 for the effect of payment for noncovered services under the guarantee of payment provision. See 4l5.3.C for the utilization of covered charges in the PRICER outlier calculation: A. Services Which May Be Billed to Beneficiaries.--All services for which charges to beneficiaries and other persons may be made under 4l5.3 are noncovered services. The charges for them are shown as noncovered charges on the inpatient billing form. B. Medically Unnecessary Outlier Days for Which You May Not Charge.--When a hospital stay exceeds the day outlier threshold, there may be days which are treated as noncovered because they are medically unnecessary, but for which you may not charge the beneficiary under 4l5.3.C. Determine these days as follows: o The number of days in excess of the day outlier threshold and pamelor.
Plasma profiles are characterized by multiple plasma concentration peaks when nateglinide is administered under fasting conditions. 375 Hudson Street, New York, NY 10014 Phone: 212-463-3400 Fax: 212-463-3590 E-mail: SWelch saatchiny Web: saatchihealthcare Founded: 1944 Parent company: Publicis Healthcare Communications Group, New York, N.Y. Officers: Michael J. Trepicchio, chairman of the board president; EVPs: Sam Welch, Dave Marek, Paul Nani, Ed Nathan, Martha Crane, Dobbi Massey, Chas Vilante, Jim Berlanti and orap.
Research & Health 23 3 ; : 187196, 1999. 19 ; Anton, R.F.; Drobes, D.J.; and George, M.S. Use of functional MRI to evaluate brain activity during alcohol cue exposure in alcoholics: Relationship to craving. Alcoholism: Clinical and Experimental Research 25 Suppl. 5 ; : 107S 108S, 2001. ; George, M.S.; Anton, R.F.; Bloomer, C.; et al. Activation of prefrontal cortex and anterior thalamus in alcoholic subjects on exposure to alcohol-specific cues. Archives of General Psychiatry 58 4 ; : 345352, 2001. 21 ; Marlatt, G.A.; Barrett, K.; and Daley, D.C. Relapse prevention. In: Galanter, M., and Kleber, H.D., eds. American Psychiatric Press Textbook of Substance Abuse Treatment. 2d ed. Washington, DC: the Association, 1999. pp. 353366. 22 ; Finney, J.W.; Noyes, C.A.; Coutts, A.I.; and Moos, R.H. Evaluating substance abuse treatment process models: I. Changes on proximal outcome variables during 12-step and cognitive-behavioral treatment. Journal of Studies on Alcohol 59: 371380, 1998. ; Volpicelli, J.R.; Alterman, A.I.; Hayashida, M.; and O'Brien, C.P. Naltrexone in the treatment of alcohol dependence. Archives of General Psychiatry 49: 876880, 1992. ; O'Malley, S.S.; Jaffe, A.J.; Chang, G.; Schottenfeld, R.S.; Meyer, R.E.; and Rounsaville, B. Naltrexone and coping skills therapy for alcohol dependence: A controlled study. Archives of General Psychiatry 49: 881887, 1992. ; Anton, R.F.; Moak, D.H.; Waid, L.R.; et al. Naltrexone and cognitive behavioral therapy for the treatment of outpatient alcoholics: Results of a placebo-controlled trial. American Journal of Psychiatry 156 11 ; : 17581764, 1999. 26 ; Kranzler, H.R. Pharmacotherapy of alcoholism: Gaps in knowledge and opportunities for research. Alcohol & Alcoholism 35 6 ; : 537 547, 2000. ; Sinclair, J.D. Evidence about the use of naltrexone and for different ways of using it in the treatment of alcoholism. Alcohol & Alcoholism 36 1 ; : 210, 2001. 28 ; Heinl, P.; Alho, H.; Kiianmaa, K.; et al. Targeted use of naltrexone without prior detoxification in the treatment of alcohol dependence: A factorial double-blind, placebo-controlled trial. Journal of Clinical Psychopharmacology 21 3 ; : 287292, 2001. 29 ; Kranzler, H.; Tennen, H.; Blomqvist, O.; et al. Targeted naltrexone for early problem drinkers. Alcohol: Clinical and Experimental Research 25 Suppl. 5 ; : 144A, 2001. 30 ; Mason, B.J., and Ownby, R.L. Acamprosate for the treatment of alcohol dependence: A review of double-blind, placebo-controlled trials. CNS Spectrums 5 2 ; : 5869, 2000. 31 ; Boening, J.A.L.; Lesch, O.M.; Spanagel, R.; Wolfgramm, J.; Narita, M.; Sinclair, D.; Mason, B.J.; and Wiesbeck, G.A. Pharmacological relapse prevention in alcohol dependence: From animal models to clinical trials. Alcoholism: Clinical and Experimental Research 25 Suppl. 5 ; : 127S131S, 2001. 32 ; Mason, B.J. Results of the multicenter study of acamprosate in the treatment of alcoholism. Biological Psychiatry 49 8 Suppl. ; : 77S, 2001. All material contained in the Alcohol Alert is in the public domain and may be used or reproduced without permission from NIAAA. Citation of the source is appreciated, for example, pcos. Purpose : To evaluate the frequency and extent of angle recession in hyphema following blunt fireworks eye injuries. Design : Case series. Participants: Twenty-six patients with unilateral hyphema due to Iranian fireworks CharshanbeSoori ; , 4-6 weeks following injury. Methods: Two examiners independently performed gonioscopy on both eyes of the participants. Main outcome measures : The frequency and extent of angle recession. Results: The age range was 7-50 median: 18 ; years. Twenty-one 81% ; of the cases were male. The left eye was involved in twenty 77% ; of the cases. The proportion of gross to microscopic hyphema was 1: Sixteen cases 58% ; had angle recession with a mean extension of 6 clock hours ranged from 45 to 360 degrees ; . The entire firework agents-related recessions were caused by `Narenjak' a home-made explosive device P value: 0.012 ; . Recession was predictable through a model R-square: 0.607, P value: 0.003 ; : young adult age group, male gender, use of Narenjak, history of happening to be closer to the explosion focus, and a more severe hyphema were the associations. P values were respectively 0.098, 0.096, 0.051, and 0.014 ; . Conclusion: Angle recession proved to be very common and extensive among the patients with hyphema injured in Charshanbe-Soori fireworks. The modifiable risk factors were the distance from the focus of explosion and the type of the firework used. Severity of the associated hyphema can alert the clinician for the presence of recession. References : 1. Capao Filipe JA, Fernandes VL, Barros H, Falcao-Reis F, Castro-Correia J. Soccer-related ocular injuries. Arch Ophthalmol 2003 May; 121 5 ; : 687-694. 2. Filipe JA, Barros H, Castro-Correia J. Sports-related ocular injuries. A three-year follow-up study. Ophthalmology 1997 Feb; 104 2 ; : 313-318. 3. Salmon JF, Mermoud A, Ivey A, Swanevelder SA, Hoffman M. The detection of post-traumatic angle recession by gonioscopy in a populationbased glaucoma survey. Ophthalmology 1994 Nov; 101 11 ; : 1844-1850. 4. Driver PJ, Cashwell LF, Yeatts RP. Airbagassociated bilateral hyphemas and angle recession. J Ophthalmol 1994 Aug 15; 118 2 ; : 250251. 5. Burke MJ, Sanitato JJ, Vinger PF, Raymond LA, Kulwin DR. Soccerball-induced eye injuries. JAMA 1983 May 20; 249 19 ; : 2682-2685 and pimozide.
Figure 1--Mean plasma insulin concentrations SEM ; after treatments with 120 mg nateglin8de NAT ; , 0.5 and 2 mg repaglinide REP ; , and placebo administered 10 min preprandial. P 0.05 for the following treatment comparisons: * 120 mg NAT vs. placebo; * 2 mg REP vs. placebo; 120 mg NAT vs. 0.5 mg REP; and 120 mg NAT vs. 2 mg REP comparisons between 0.05 mg repaglinide and placebo not shown. Once the medicine is approved, a wider range of people begin taking the medicine and for longer periods of time and orinase. 10 improvement of insulin resistance by a new insulin secretagogue, nateglinide-analysis based on the homeostasis model.

The school nurse RN or LPN ; may administer oxygen through a nasal cannula or mask. Use of a tracheostomy collar may require a registered school nurse or respiratory therapist with training, depending on the care needs of the student with a tracheostomy and as specified in the student's individualized health care plan. Any school personnel who have regular contact with a student who requires oxygen should receive general training covering the student's specific needs, potential problems and implementation of the established emergency plan. The level of 1-AR in AChE thalamus was about fourfold higher than in wild-type counterparts. Other studied brain regions did not display any differences between two genotypes. It is supposed that AChE - mice are able to live due to multiple adaptation changes in their phenotype. Cholinergic adaptations were described 4, 5 ; in the past. Unfortunately only little attention has been paid to the non-cholinergic changes in AChE CNS. We show in this work that -adrenoceptors are significantly changed in the CNS of AChE mice while these changes are brain-region specific. 1 ; J. Hartmann et al., XII ISCM, Alicante, Spain, 2005; P006, 74, 2 ; M. Teplick et al., Physiol. Res. 2004; 53, 4, ; W. Xie et al., Pharmacol. Exp. Ther. 2000; 293, 896-902, ; L.A. Volpicelli-Daley et al., Ann. Neurol. 2003; 53, 788-796, ; B. Li et al., Pharmacol Biochem Behav. 2003; 74, 977-86, Supported by U.S. Army Medical Research and Materiel Command Grant DAMD17-01-2-0036 to O.L. UNMC Eppley Cancer Center Support Grant P30CA36727-19; by Grant Agency of Charles University GAUK 36 04 to J.M. by Ministry of Education of Slovak Republic VEGA 1 2271 05 to A.H. ; and by Slovak-Czech Grant SK-87 CZ-112.

The postprandial insulinotropic effects of nateglinide are more rapid than those of repaglinide and more rapid and greater than those of glibenclamide glyburide ; , while producing less prolonged insulin exposure and less risk of delayed hypoglycaemia.

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