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Haematology and biochemistry. We used intraoesophageal 24-hour pH measurement, ultrasonography and gastric scintigraphy. Results: 24 hour pH measurement established the diagnosis of GOR in 12 26 patients 46.1% ; and Doppler sonography in 15 26 57.6% ; . In nine patients GOR was verified by both methods. In the latter gastric scintigraphy was subsequently performed and found positive in four patients. From the control group only one had established GOR verified by the above processes. No patient was found with mild GOR, although from our sample 46.6% ; were found with moderate and 58.4% ; with severe GOR. In the spastic types of cerebral palsy diplegia, tetraplegia, hemiplegia ; GOR was found in 46.5% whereas in the other types in 35.3%. The most frequently associated problems with GOR were malnutrition, anaemia, iron deficiency, and infections of the lower respiratory tract. Conclusion: In conclusion according to our results 24hour pH measurement evaluation is most accurate and of high specificity in over 90% of syptomatic patients. It could be used as a screening test coupled with Doppler sonography for unveiling GOR in high risk patients.
The same as the prescriber's social security number needed on out patient prescriptions for cs the national durg code ndc ; is usually rquired for purchasing of pharmaceutical products, especially multisource products, for example, keflex dosing. SCF forms, the membrane bound and the free, soluble one, which originates from the membrane bound form dissociated by proteolytic proteases and has significant biological effects 14 ; . Mast cells do not produce SCF. On the surface membrane of mast cells there are c-Kit proteins regarded as SCF receptors, in fact they are their ligands 15, 16 ; . The above-mentioned receptors of mast cells are product of c-Kit protooncogene being a component of the structural gene known as exon 6 13, 17 ; . Based on comprehensive studies 18, 19 ; , SCF is an important factor with the impact on the number, phenotype and function of mast cells in tissues of healthy people. In mastocytosis SCF mutations, or disturbed metabolism, are to be taken into consideration 16, 20 ; . As mast cells together with eosinophils, monocytes, neutrophils and basophils ; derive from CD34 positive multipotent germinal hematopoietic cells in the bone marrow, the pathologic SCF synthesis can under certain circumstances influence even the bone marrow stem cells. It can also be one of the explanations why the diseases of hematopoietic system occur in mastocytosis patients as an accompanying phenomenon. The spontaneous regression of mastocytosis may depend on endocrinologic, immunologic, biochemical, or other processes occurring in human organism during the period of adolescence. The mentioned development may influence the mast cells evolution from expression of c-Kit protooncogene up to production of SCF 21 ; . In this study SCF was not identified, but the expressivity on mast cells of CD34 was the same as it was the first biopsy or repeated dermal biopsies through or after the treatment of mastocytosis. Therapeutical effects on mast cell infiltrates of mastocytosis as observed by Kurosawa et al 5 ; and as confirmed by our study are, also to be taken into consideration. Dermal mast cells infiltrates were through the treatment period less expressed and changed from band-like type to type of vessel- unit or from type of vessel-unit more then 55 m to vessel-unit less than 55m. 13. British Thoracic Society. British guidelines on asthma management. Thorax 1997; 52 Suppl 1 ; : 1S-21S. 14. SPSS Statistical package for social sciences ; [computer program]. Version 10. Chicago, Ill: SPSS, 2000. 15. N4, NVivo [computer program]. Version 1. Melbourne: Qualitative Solutions & Research International, 2000. 16. Message S, Johnston S. Viruses in asthma. Br Med Bull 2002; 61: 29-43. Johnston S, Pattemore P, Sanderson G, et al. The relationship between upper respiratory tract infections in adults requiring hospitalisation for asthma: a time trend analysis. J Respir Crit Care Med 1996; 154: 654-660. Reddell H, Ware S, Marks G, et al. Differences between asthma exacerbations and poor asthma control. Lancet 1999; 353: 364-369. Ruffin R, Wilson D, Smith B, et al. Prevalence, morbidity and management of adult asthma in South Australia. Immunol Cell Biol 2001; 79: 191-194. Wilson D, Adams R, Appleton S, et al. Prevalence of asthma and asthma action plans in South Australia: population surveys from 1990 to 2001. Med J Aust 2003; 178: 483-485. Matheson M, Wicking J, Raven J, et al. Asthma management: how effective is it in the community? Int Med J 2002; 32: 451-456. Asthma 3 + visit plan 2002. Canberra: Commonwealth Department of Health and Ageing. Available at: health.gov.au pq asthma 3visitpln accessed Nov 2003 ; . 23. Glasgow N, Ponsonby A, Yates R, et al. Proactive asthma care in childhood: general practice based randomised controlled trial. BMJ 2003; 327: 659-663. Received 9 Jul 2003, accepted 20 Oct 2003 and nifedipine.

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